认知 — 思想的食粮

就其体积而言,大脑的能量需求十分巨大,人体 22% 的氧气消耗量是为了供给这个仅占成年人体重约 2% 的器官 [1]。大脑需要大量的氧气来将葡萄糖转化为能量 [1]。健康成年人大脑能量需求的 97% 由葡萄糖满足,其余则来自酮 [1、2]。然而,随着年龄的增长,酮可以提供超过 60% 的大脑能量需求 [3]。大脑对葡萄糖的摄取量会随年龄增长而减少,人类研究显示,老年受试者的葡萄糖摄取量比非老年受试者减少 10-15% [4-7]。进一步的证据显示,阿尔茨海默症患者的葡萄糖摄取量减少了 20-25% [4-7]。有人提出,大脑对葡萄糖的使用和/或摄取可能在临床认知功能下降之前很久就已开始减少,尤其是有阿尔茨海默症遗传倾向的人 [7]。学习和记忆也可能受到葡萄糖缺乏状况的影响 [8-10]。

特色产品:Captex® 8000(高 C8 含量 MCT)

ABITEC 的 Captex 中链甘油三酯 (MCT) 分为多个等级,每个等级都具有独特特性,适合不同配方。我们的 Captex 8000 MCT 含有 98% 的 C8 酰基。

MCT 是通常含有 8-12 个碳原子的脂肪。每种 MCT 的 C8-C12 含量可能不同。CAPTEX 8000 具有高 C8 酰基含量,含碳量低于具有更长酰基的脂肪(甘油三酯)。这是非常重要的,因为研究表明,与碳链较长的 MCT 相比,摄入高 C8 的 MCT 显著增加了酮的产生 [11,12]。Cunnane 等人最近的一项研究显示,源于此类 MCT 的 C8 脂肪酸在 8 小时内增加的血酮量是 C10 的 3 倍以上 [13]。

ABITEC 的 Captex 8000 可以单独使用,也可加入多种不同配方中,例如:

  • 即饮 (RTD) 饮料
  • 能量棒
  • 能量胶
  • 能量弹
  • 咖啡奶精

产品名称

化学名称 辛酸 (C8) 含量 (%) 癸酸 (C10) 含量 (%) CAS 编号
Captex 8000  三辛酸甘油酯 98 <1 538-23-8

相关产品

CAPTEX®

中链甘油三酯 (MCT) 是身体和大脑容易获得的一种能量来源,能够增加酮的生成,有助于为大脑的运行提供燃料(能量)。

此外,其还具有以下特点:

  • 犹太洁食和清真食品
  • 易于融入多种剂型,如功能性饮料、能量棒、软糖、能量胶、粉末等。
  • 生产原料不含生物工程成分(非转基因)
  • 容易获得,有助于认知功能、肌肉恢复、体重健康 [14-16]
  • 我们的 Nutri Sperse® 产品系列提供粉末形态
  • 优异的稳定性,保质期 24 - 36 个月

提供多种可持续原材料选择 

椰子、无棕榈及 RSPO 选择

产品名称

化学名称 辛酸 (C8) 含量 (%) 癸酸 (C10) 含量 (%) CAS 编号
Captex 300 三辛酸/三癸酸甘油酯 ~70 ~30  65381-09-1
Captex 355 三辛酸/三癸酸甘油酯 ~55 ~35 65381-09-1、
73398-61-5
Captex 1000  三癸酸甘油酯 <1 98 621-71-6

访问我们的资源中心

NUTRI SPERSE®

NUTRI SPERSE 脂质粉末可传递多种健康油品中所含高达 73% 的脂肪量,包括中链甘油三酯 (MCT)、菜籽油和高油酸葵花籽油。我们所有的油品均为低反式脂肪、非氢化且符合犹太洁食和清真食品标准。它们在各种冷热水应用中均具有良好的分散性,并可为不适用液体油的产品提供光滑细腻的口感和更优的功能性。

此外,它们还具有以下特点:

  • 提供乳制品基料和非乳制品基料*
  • 含有 MCT,能增加能量供应并实现可持续的能量供应
  • 用于促进运动后恢复 
  • 保质期长达 24 个月

*Nutri Sperse 粉末基料中的碳水化合物和蛋白质经专门选择,以提供营养功能,例如由慢至快的能量产生。

产品名称

基料

油品类型 

 %

Nutri Sperse MCT 50 非转基因麦芽糊精 MCT 50%
Nutri Sperse MCT 70 脱脂奶粉 MCT 70%

访问我们的资源中心

Related References

1. Cahill, G.F., Jr., Fuel metabolism in starvation. Ann Rev Nutr, 2006. 26: p. 1-22.
2. Dahlquist, G. and B. Persson, The rate of cerebral utilization of glucose, ketone bodies, and oxygen: a comparative in vivo study of infant and adult rats. Pediatr Res, 1976. 10(11): p. 910-7.
3. Owen, O.E., et al., Brain metabolism during fasting. J Clin Invest, 1967. 46(10): p. 1589-95.
4. Blennow, K., M.J. de Leon, and H. Zetterberg, Alzheimer’s disease. Lancet, 2006. 368(9533): p. 387-403.
5. Mosconi, L., et al., Hypometabolism exceeds atrophy in presymptomatic early-onset familial Alzheimer’s disease. J Nuclear Med, 2006. 47(11): p. 1778-86.
6. Ogawa, M., et al., Altered energy metabolism in Alzheimer’s disease. J Neurol Sci, 1996. 139(1): p. 78-82.
7. Cunnane, S., et al., Brain fuel metabolism, aging, and Alzheimer’s disease. Nutrition, 2011. 27(1): p. 3-20.
8. McNay, E.C., T.M. Fries, and P.E. Gold, Decreases in rat extracellular hip pocampal glucose concentration associated with cognitive demand during a spatial task. Proc Natl Acad Sci U S A, 2000. 97(6): p. 2881-5.
9. McNay, E.C. and P.E. Gold, Age-related differences in hippocampal extra cellular fluid glucose concentration during behavioral testing and following systemic glucose administration. J Gerontol A Biol Sci Med Sci, 2001. 56(2): p. B66-71.
10. McNay, E.C., R.C. McCarty, and P.E. Gold, Fluctuations in brain glucose con centration during behavioral testing: dissociations between brain areas and between brain and blood. Neurobiol Learn Mem, 2001. 75(3): p. 325-37.
11. McGarry, J. D., & Foster, D. W. (1971). The Regulation of Ketogenesis from Octanoic Acid The Role of the Tricarboxylic Acid Cycle and Fatty Acid Synthesis. Journal of Biological Chemistry246(4), 1149-1159.
12. Miles, J. M., Haymond, M. W., Nissen, S. L., & Gerich, J. E. (1983). Effects of free fatty acid availability, glucagon excess, and insulin deficiency on ketone body production in postabsorptive man. Journal of Clinical Investigation71(6), 1554.
13. Vandenberghe, C., St-Pierre, V., Pierotti, T., Fortier, M., Brodeur-Dubreuil, C., Cunnane, S.C. (2016). Acute plasma ketone response to coconut oil alone or in combination with different medium chain triglycerides. ISSFAL Congress, Banf Canada, Poster Presentation.
14. St. Onge, M.P. , Jones, J.H. (2002) Physiological Effects of Medium Chain triglycerides: Potential Agents in the Prevention of Obesity, J. Nutr. 132: 329-332
15. Bach AC and Babayan VK. Medium-chain triglycerides: an update. The American journal of clinical nutrition 1982;36(5):950-62.
16. Heydinger JA and Nakhasi DK. Medium Chain Triacylglycerols. J Food Lipids 1996;3:251-257.

Legal Disclaimer

CAPTEX® is aregistered trademark of ABITEC Corporation. All information and statements given on this website are believed to be accurate at the time of publication. However, neither ABITEC Corporation nor any of their affiliates make any representations or warranty with respect thereto, including, but not limited to, any results obtained in the processing of the products by customers or any third party. All information and statements are intended for persons having the required skill and know-how and do not relieve the customer or user from verifying the suitability of information and statements given for a specific purpose prior to use of the products. It is entirely the obligation of the customer or user to comply with applicable laws and regulations, and also with all patent or other intellectual property rights of third parties. ABITEC CORPORATION EXPRESSELY DISCLAIMS ANY REPRESENTATIONS OR WARRANTIES OF ANY KIND, WHETHER EXPRESSED OR IMPLIED, AS TO THE ACCURACY, CURRENCY, COMPLETENESS AND/OR THE MERCHANTABILITY OR FITNESS OF A PARTICULAR PURPOSE OF ANY INFORMATION CONTAINED ON THIS WEBSITE AND/OR PRODUCT DESCRIBED OR PROMOTED ON THIS WEBSITE, INCLUDING WARRANTIES WITH RESPECT TO INFRINGEMENT OF ANY PATENT, COPYRIGHT, OR OTHER RIGHTS OF A THIRD PARTY. We reserve the right to change product specifications and specified properties of the products without prior notice. NUTRI SPERSE is a registered trademark of AB Mauri Food Inc.  Used under license.